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CRISPR/Cas9-mediated inactivation of miR-34a and miR-34b/c in

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miR-34a induces senescence-associated growth arrest in liver

miR-34a exerts as a key regulator in the dedifferentiation of

miR-34a exerts as a key regulator in the dedifferentiation of

R-34 family miRNAs mediate growth arrest in a variety of cell types.a

Activation of miR-34a impairs autophagic flux and promotes cochlear cell death via repressing ATG9A: implications for age-related hearing loss

The miR-34 family in cancer and apoptosis

Targeting miRNA by CRISPR/Cas in cancer: advantages and challenges

Biomedicines, Free Full-Text

CRISPR/Cas9-mediated inactivation of miR-34a and miR-34b/c in

Frontiers NOP58 induction potentiates chemoresistance of colorectal cancer cells through aerobic glycolysis as evidenced by proteomics analysis